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Schnappinger, Dirk
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Freire DMendes
,
Gutierrez C
,
Garza-Garcia A
,
Grabowska AD
,
Sala AJ
,
Ariyachaokun K
,
Panikova T
,
Beckham KSH
,
Colom A
,
Pogenberg V
et al.
. 2019.
An NAD Phosphorylase Toxin Triggers Mycobacterium tuberculosis Cell Death.
.
Mol Cell. 73(6):1282-1291.e8.
M
Shi S
,
Nathan C
,
Schnappinger D
,
Drenkow J
,
Fuortes M
,
Block E
,
Ding A
,
Gingeras TR
,
Schoolnik G
,
Akira S
et al.
. 2003.
MyD88 primes macrophages for full-scale activation by interferon-gamma yet mediates few responses to Mycobacterium tuberculosis.
.
J Exp Med. 198(7):987-97.
Williams KJ
,
Boshoff HI
,
Krishnan N
,
Gonzales J
,
Schnappinger D
,
Robertson BD
. 2011.
The Mycobacterium tuberculosis β-oxidation genes echA5 and fadB3 are dispensable for growth in vitro and in vivo.
.
Tuberculosis (Edinb). 91(6):549-55.
Ehrt S
,
Schnappinger D
. 2007.
Mycobacterium tuberculosis virulence: lipids inside and out.
.
Nat Med. 13(3):284-5.
Lin K
,
O'Brien KM
,
Trujillo C
,
Wang R
,
Wallach JB
,
Schnappinger D
,
Ehrt S
. 2016.
Mycobacterium tuberculosis Thioredoxin Reductase Is Essential for Thiol Redox Homeostasis but Plays a Minor Role in Antioxidant Defense.
.
PLoS Pathog. 12(6):e1005675.
Gouzy A
,
Larrouy-Maumus G
,
Bottai D
,
Levillain F
,
Dumas A
,
Wallach JB
,
Caire-Brandli I
,
de Chastellier C
,
Di Wu T-
,
Poincloux R
et al.
. 2014.
Mycobacterium tuberculosis exploits asparagine to assimilate nitrogen and resist acid stress during infection.
.
PLoS Pathog. 10(2):e1003928.
Ehrt S
,
Schnappinger D
. 2009.
Mycobacterial survival strategies in the phagosome: defence against host stresses.
.
Cell Microbiol. 11(8):1170-8.
Stephanou NC
,
Gao F
,
Bongiorno P
,
Ehrt S
,
Schnappinger D
,
Shuman S
,
Glickman MS
. 2007.
Mycobacterial nonhomologous end joining mediates mutagenic repair of chromosomal double-strand DNA breaks.
.
J Bacteriol. 189(14):5237-46.
Ehrt S
,
Rhee K
,
Schnappinger D
. 2015.
Mycobacterial genes essential for the pathogen's survival in the host.
.
Immunol Rev. 264(1):319-26.
Ehrt S
,
Schnappinger D
,
Rhee KY
. 2018.
Metabolic principles of persistence and pathogenicity in Mycobacterium tuberculosis.
.
Nat Rev Microbiol. 16(8):496-507.
Vandal OH
,
Pierini LM
,
Schnappinger D
,
Nathan CF
,
Ehrt S
. 2008.
A membrane protein preserves intrabacterial pH in intraphagosomal Mycobacterium tuberculosis.
.
Nat Med. 14(8):849-54.
Shi C
,
Geders TW
,
Park SWoong
,
Wilson DJ
,
Boshoff HI
,
Abayomi O
,
Barry CE
,
Schnappinger D
,
Finzel BC
,
Aldrich CC
. 2011.
Mechanism-based inactivation by aromatization of the transaminase BioA involved in biotin biosynthesis in Mycobaterium tuberculosis.
.
J Am Chem Soc. 133(45):18194-201.
L
Johnson EO
,
LaVerriere E
,
Office E
,
Stanley M
,
Meyer E
,
Kawate T
,
Gomez JE
,
Audette RE
,
Bandyopadhyay N
,
Betancourt N
et al.
. 2019.
Large-scale chemical-genetics yields new M. tuberculosis inhibitor classes.
.
Nature. 571(7763):72-78.
I
Ehrt S
,
Schnappinger D
. 2003.
Isolation of plasmids from E. coli by boiling lysis.
.
Methods Mol Biol. 235:79-82.
Ehrt S
,
Schnappinger D
. 2003.
Isolation of plasmids from E. coli by alkaline lysis.
.
Methods Mol Biol. 235:75-8.
Schnappinger D
,
Ehrt S
. 2006.
Introduction: genomic approaches in infectious diseases.
.
Microbes Infect. 8(6):1611-2.
Voskuil MI
,
Schnappinger D
,
Visconti KC
,
Harrell MI
,
Dolganov GM
,
Sherman DR
,
Schoolnik GK
. 2003.
Inhibition of respiration by nitric oxide induces a Mycobacterium tuberculosis dormancy program.
.
J Exp Med. 198(5):705-13.
Puckett S
,
Trujillo C
,
Eoh H
,
Marrero J
,
Spencer J
,
Jackson M
,
Schnappinger D
,
Rhee K
,
Ehrt S
. 2014.
Inactivation of fructose-1,6-bisphosphate aldolase prevents optimal co-catabolism of glycolytic and gluconeogenic carbon substrates in Mycobacterium tuberculosis.
.
PLoS Pathog. 10(5):e1004144.
Gandotra S
,
Schnappinger D
,
Monteleone M
,
Hillen W
,
Ehrt S
. 2007.
In vivo gene silencing identifies the Mycobacterium tuberculosis proteasome as essential for the bacteria to persist in mice.
.
Nat Med. 13(12):1515-20.
Klotzsche M
,
Ehrt S
,
Schnappinger D
. 2009.
Improved tetracycline repressors for gene silencing in mycobacteria.
.
Nucleic Acids Res. 37(6):1778-88.
Wescott HH
,
Zuniga ES
,
Bajpai A
,
Trujillo C
,
Ehrt S
,
Schnappinger D
,
Roberts DM
,
Parish T
. 2018.
Identification of Enolase as the Target of 2-Aminothiazoles in .
.
Front Microbiol. 9:2542.
G
Puckett S
,
Trujillo C
,
Wang Z
,
Eoh H
,
Ioerger TR
,
Krieger I
,
Sacchettini J
,
Schnappinger D
,
Rhee KY
,
Ehrt S
. 2017.
Glyoxylate detoxification is an essential function of malate synthase required for carbon assimilation in Mycobacterium tuberculosis.
.
Proc Natl Acad Sci U S A. 114(11):E2225-E2232.
Santangelo Mde la Paz
,
Gest PM
,
Guerin ME
,
Coinçon M
,
Pham H
,
Ryan G
,
Puckett SE
,
Spencer JS
,
Gonzalez-Juarrero M
,
Daher R
et al.
. 2011.
Glycolytic and non-glycolytic functions of Mycobacterium tuberculosis fructose-1,6-bisphosphate aldolase, an essential enzyme produced by replicating and non-replicating bacilli.
.
J Biol Chem. 286(46):40219-31.
Marrero J
,
Rhee KY
,
Schnappinger D
,
Pethe K
,
Ehrt S
. 2010.
Gluconeogenic carbon flow of tricarboxylic acid cycle intermediates is critical for Mycobacterium tuberculosis to establish and maintain infection.
.
Proc Natl Acad Sci U S A. 107(21):9819-24.
Schnappinger D
. 2007.
Genomics of host-pathogen interactions.
.
Prog Drug Res. 64:311,313-43.
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